Download Adipose Tissue and Adipokines in Health and Disease by Saverio Cinti MD (auth.), Giamila Fantuzzi PhD, Theodore PDF

By Saverio Cinti MD (auth.), Giamila Fantuzzi PhD, Theodore Mazzone MD (eds.)

This e-book provides a entire survey of adipose tissue, its physiological services, and its function in disorder. the amount spans the whole diversity of adipose tissue stories, from easy anatomical and physiological examine to epidemiology and medical stories. The authors are individual researchers, clinicians and epidemiologists. they've got integrated groundbreaking fresh stories into conventional types of adipose tissue homes.

Chapters on famous houses of adipokines leptin and adiponectin are complemented by way of an creation to a unique view of adipose tissue as a dynamic organ that regulates systemic substrate availability and metabolism, in addition to various different discrete services. This novel idea is improved.

Worldwide tendencies in weight problems are mentioned from an evolutionary standpoint and factors of the present weight problems epidemic are postulated. also, researchers and clinicians learn the organization and strength function of adipose tissue in illness mediation and provide epidemiological facts. This quantity concludes with a considerate and cutting edge dialogue of varied techniques to inducing and maintaining weight-loss in overweight

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Extra info for Adipose Tissue and Adipokines in Health and Disease

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These mice do not develop obesity and have a reduced fat mass (100,101). They always have a marked basal lipolysis and a response of lipolysis to G-adrenergic stimuli (100–102). These findings suggested than another lipase was present and active in the absence of HSL. The finding that DAG accumulates in adipocytes of these mice (103) suggested that this lipase had a preference for the hydrolysis of TAG, and was rate-limiting for this first step of lipolysis, whereas HSL was limiting for the hydrolysis of DAG.

De Matteis R, Ricquier D, Cinti S. L. J Neurocytol 1998;27(12):877–886. Giordano, A, Frontini A, Murano I, et al. J Histochem Cytochem 2005;53:679–687. Cannon B, Nedergaard J. Biochem Soc Trans 1986;14:233–236. Cinti S. Ital J Anat Embryol 1999;104:37–51. Giordano A, Morroni M, Carle F, et al. J Cell Sci 1998;111(Pt 17):2587–2594. Cannon B, Hedin A, Nedergaard J. FEBS Lett 1982;150:129–132. Trayhurn P, Nicholls DG, Brown Adipose Tissue. Edward Arnold, London, 1986. 18 15. 16. 17. 18. 19. 20. 21.

Between that of “white” mitochondria and that of “brown” mitochondria. We think that this type of multilocular adipocyte rich in mitochondria is the morphological equivalent of a white adipocyte transdifferentiating into a brown adipocyte. Some of these cells, also found in white areas of the adipose organ of cold-exposed animals, are immunoreactive for UCP1. Of note, 80 to 95% of these cells are BrdU negative, suggesting that their development is independent from mitotic processes (42,43). Recently, it has been shown that Sv129 mice are quite resistant to obesity and diabetes in comparison with B6 mice (44).

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